PUBLIC HEALTH ASSESSMENT
LOXAHATCHEE NURSERY
PALM CITY, MARTIN COUNTY, FLORIDA
| ATSDR | Agency for Toxic Substances and Disease Registry |
|---|---|
| bls | below land surface |
| BHC | Hexachlorocyclohexane |
| DDD | 1,1-dichloro-2,2-bis(p-chlorophenyl)ethane |
| DDE | 1,1-dichloro-2,2-bis(p-chlorophenyl)ethylene |
| DDT | 1,1,1-trichloro-2,2-bis(p-chlorophenyl)ethane |
| CLHA | Child Longer Term Health Advisory |
| CREG | Cancer Risk Evaluation Guide |
| EMEG | Environmental Media Evaluation Guide |
| EPA | Environmental Protection Agency |
| FDEP | Florida Department of Environmental Protection |
| HRS | Department of Health and Rehabilitative Services |
| IARC | International Agency for Research on Cancer |
| kg | kilograms |
| L | liter |
| LTHA | Lifetime Health Advisory |
| MCHD | Martin County Health Department |
| MCL | Maximum Contaminant Level |
| MCPHU | Martin County Public Health Unit |
| mg | milligrams |
| MRL | Minimal Risk Level |
| NOAEL | No Observed Adverse Effect Level |
| NTP | National Toxicology Program |
| PCBs | Polychlorinated Biphenyls |
| ppb | parts per billion |
| ppm | parts per million |
| RBCn | Risk-Based Concentration (noncancer) |
| RMEG | Reference Dose Media Evaluation Guide |
| SVOCs | Semi-Volatile Organic Compounds |
| µg | micrograms |
| VOCs | Volatile Organic Compounds |

Figure 1. March 1998 Sampling Event Location Map -- Draft

Figure 2. Demographic Information
| Pathway Name | Source | Contaminants | Environmental Medium | Point of Exposure | Route of Exposure | Exposed Population | Time Exposed |
|---|---|---|---|---|---|---|---|
| Groundwater | Potentially the Nursery | See Table 4 (private well data) | Off-Site Groundwater | Water from private drinking wells | Ingestion, Inhalation, Dermal | Residents with private drinking wells | Past, Current, Future |
| Pathway Name | Source | Contaminants | Environmental Medium | Point of Exposure | Route of Exposure | Exposed Population | Time Exposed |
|---|---|---|---|---|---|---|---|
| On-Site Soil | Nursery | See Tables 5 and 6 | On-Site Soil | Direct contact with on-sitesoils | Dermal, IncidentalIngestion | Residents living on-site; Trespassers | Past, Current, Future |
| Off-Site Soil | PotentiallytheNursery | See Table 7 | Off-Site Soil | Direct contact with off-sitesoils | Dermal, IncidentalIngestion | Residents | Past, Current, Future |
| On-Site Sediment | Nursery | See Table 8 | On-Site Sediment | Direct contact with on-site sediment in drainage ditches and the pond | Dermal, Incidental Ingestion | Trespassers | Past, |
| Off-siteSediment | PotentiallytheNursery | See Table 8 | Off-Site Sediment | Direct contact with off-sitesediment in drainageditches and the South Forkof the St. Lucie River | Dermal, IncidentalIngestion | Residents | Past, Current, Future |
| Contaminant | Concentration Range1 (ppb)2 | Year of Max, Sample ID | Comparison Value3 Value (ppb) Source | |
|---|---|---|---|---|
| Benzene | 8 | 1994, Temporary Well | 5 | MCL |
| beta-BHC | 0.025 I6 - 3.3 | 1995, MW#3 | 6 | Intermediate EMEG (child) |
| 20 | Intermediate EMEG (adult) | |||
| gamma-BHC | 0.05 | 1995, MW#3 | 0.4 | Intermediate EMEG (child) |
| 1 | Intermediate EMEG (adult) | |||
| 0.2 | MCL | |||
| delta-BHC | 0.11 (2 hits) | 1995, MW#3 | None | |
| Chlordane | 0.27 I - 200 | 1992, 1A#1A | 6 | Chronic EMEG (child) |
| 20 | Chronic EMEG (adult) | |||
| Chloromethane | 0.54 I | 1998, TW03 | 3 | LTHA |
| 400 | CLHA | |||
| 4,4 - DDD | 0.031 I; 1.49 | 1992, 1A#1A | 0.1 | CREG |
| 4,4 - DDE | 0.12 | 1992, 1A#1A | 0.1 | CREG |
| Diazinon | 4.9 | 1992, TW#2 | 2 | Intermediate EMEG (child) |
| 7 | Intermediate EMEG (adult) | |||
| 5 | CLHA | |||
| Endosulfan I | 0.12 | 1992, 1A#1A | 20 | Chronic EMEG (child)5 |
| 70 | Chronic EMEG (adult)5 | |||
| Endosulfan Sulfate | 1.46 | 1992, 1A#1A | 20 | Chronic EMEG (child)5 |
| 70 | Chronic EMEG (adult)5 | |||
| Aluminum | 220 | 1998, TW04 | 37,000 | RBCn |
| Arsenic | 3 I; 11.2 | 1998, TW04 | 3 | Chronic EMEG (child) |
| 10 | Chronic EMEG (adult) | |||
| 50 | MCL | |||
| Barium | 3.75 J4 - 20 | 1998, TW04 | 700 | RMEG (child) |
| 2,000 | RMEG (adult) | |||
| Cadmium | 0.05 I; 0.1 I | 1998, TW03 | 7 | Chronic EMEG (child) |
| 20 | Chronic EMEG (adult) | |||
| Calcium | 30,000 J - 85,000 J | 1998, TW04 | None | |
| Chromium | 2 J - 5 I | 1998, TW03 | 100 | MCL |
| Copper | 15 J - 25 J | 1998, TW04 | 1,500 | RBCn |
| Iron | 91 - 1,600 | 1998, TW04 | 11,000 | RBCn |
| Lead | 0.58 - 4 J | 1998, TW03 | 15 | EPA Action Level |
| Magnesium | 1,000 - 4,000 | 1998, TW04 | None | |
| Manganese | 15 J - 41 | 1998, TW03 | 50 | RMEG (child) |
| 200 | RMEG (adult) | |||
| Potassium | 1,100 - 1,600 | 1998, TW04 | None | |
| Sodium | 3,000 - 30,000 | 1998, TW04 | None | |
| Zinc | 17 J - 33 | 1998, TW04 | 3,000 | Chronic EMEG (child) |
| 10,000 | Chronic EMEG (adult) | |||
Sources: ATSDR, 1996a; EPA, 1998b; Evergreen Engineering, 1996; FDEP, 1998; and McGinnes Laboratories, 1992.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppb = parts per billion.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. J = estimated value.
5. Comparison value is for Endosulfan.
6. I = value reported is less than the minimum quantitation limit, and greater than or equal to the minimum detection limit.
| Contaminant | Private Wells Concentration Range1 (ppb)2 | Private Wells Year of Max | Temporary Well Concentration in ppb (1998) | Comparison Value3 Value (ppb) Source | |
|---|---|---|---|---|---|
| Benzene | 0.31 - 1 J4 | 1998 | ND5 | 1 | CREG |
| 5 | MCL | ||||
| Bromodichloro- methane | 1.3; 3.4 | 1996 | ND | 0.6 | CREG |
| 200 | Chronic EMEG (child) | ||||
| 700 | Chronic EMEG (adult) | ||||
| Bromoform | 1.2 | 1996 | ND | 4 | CREG |
| 2,000 | Chronic EMEG (child) | ||||
| 7,000 | Chronic EMEG (adult) | ||||
| Chloroform | 0.51; 9.9 | 1996 | ND | 6 | CREG |
| 100 | Chronic EMEG (child) | ||||
| 400 | Chronic EMEG (adult) | ||||
| Chloromethane | 0.13 - 0.82 I6 | 1998 | 0.58 I | 3 | LTHA |
| 400 | CLHA | ||||
| Dibromochloro- methane | 0.54; 2.8 | 1996 | ND | 0.4 | CREG |
| 300 | Chronic EMEG (child) | ||||
| 1,000 | Chronic EMEG (adult) | ||||
| Di (2-ethylhexyl)phthalate | 3.0 | 1996 | ND | 3 | CREG |
| 200 | RMEG (child) | ||||
| 700 | RMEG (adult) | ||||
| 1,1-Dichloroethane | 0.21 | 1996 | ND | 810 | RBCn |
| 1,2-Dichloropropane | 0.1 - 330 | 1996 | ND | 900 | Chronic EMEG (child) |
| 3000 | Chronic EMEG (adult) | ||||
| 5 | MCL | ||||
| Naphthalene | 0.22; 0.37 | 1996 | ND | 200 | Intermediate EMEG (child) |
| 700 | Intermediate EMEG (adult) | ||||
| Styrene | 0.23; 0.31 | 1996 | ND | 2,000 | Intermediate EMEG (child) |
| 7,000 | Intermediate EMEG (adult) | ||||
| 100 | MCL | ||||
| 1,2,3-Trichloropropane | 1.8 - 3.6 | 1996 | ND | 60 | RMEG (child) |
| 200 | RMEG (adult) | ||||
| Aluminum | 89 | 1998 | ND | 37,000 | RBCn |
| Arsenic | ND | 1998 | 27; 28.3 | 0.02 | CREG |
| 3 | Chronic EMEG (child) | ||||
| 10 | Chronic EMEG (adult) | ||||
| Barium | 15.6 - 23 | 1998 | 2.16 J | 700 | RMEG (child) |
| 2,000 | RMEG (adult) | ||||
| Calcium | 1,500 J - 90,000 J | 1998 | 68,000 J | None | |
| Chromium | 3 J | 1998 | 2 J | 100 | MCL |
| Copper | 15 J, 25 J | 1998 | ND | 1,500 | RBCn |
| Iron | 47 - 7,200 | 1998 | 380 | 11,000 | RBCn |
| Lead | 0.4 I - 3 J | 1998 | 0.3 I | 15 | EPA Action Level |
| Magnesium | 730 - 3,200 | 1998 | 16,000 | None | |
| Manganese | 6 J - 46 | 1998 | 16 | 50 | RMEG (child) |
| 200 | RMEG (adult) | ||||
| Potassium | 210 - 1,400 | 1998 | 1,800 | None | |
| Sodium | 3,400 - 160,000 | 1998 | 15,000 | None | |
| Vanadium | 3 J | 1998 | ND | 30 | Intermediate EMEG (child) |
| 300 | Intermediate EMEG (adult) | ||||
| Zinc | 38 - 210 | 1998 | ND | 3,000 | Chronic EMEG (child) |
| 10,000 | Chronic EMEG (adult) | ||||
Sources: EPA, 1998b; EPA, 1998f; FDEP, 1998; HRS, 1996b; and HRS, 1996c.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppb = parts per billion.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. J = estimated value.
5. ND = material was analyzed for but not detected (value below the minimum quantitation limit).
6. I = value reported is less than the minimum quantitation limit, and greater than or equal to the minimum detection limit.
| Contaminant | Concentration Range1 (ppm)2 | Year of Max, Sample ID | Comparison Value 3 Value (ppm) Source | |
|---|---|---|---|---|
| Chlordane | 0.015 - >1.0 | 1992, #1Soil | 1 | Chronic EMEG (pica child) |
| 30 | Chronic EMEG (child) | |||
| beta BHC | 0.0052 | 1992, #1Soil | 0.6 | Intermediate EMEG (pica child) |
| 20 | Intermediate EMEG (child) | |||
| gamma BHC | 0.0038 | 1992, #1Soil | 0.08 | Intermediate EMEG (pica child) |
| 2 | Intermediate EMEG (child) | |||
| delta BHC | 0.0045 | 1992, #1Soil | None | |
| DDE | 0.012 - 0.072 | 1996, West | 2 | CREG |
| DDD | 0.015 - 0.030 | 1996, West | 3 | CREG |
| DDT | 0.0062; 0.011 | 1996, West | 1 | RMEG (pica child) |
| 30 | RMEG (child) | |||
| Dieldrin | 0.013 | 1996, West | 0.1 | Chronic EMEG (pica child) |
| 3 | Chronic EMEG (child) | |||
| Endosulfan II | 0.0103 | 1992, #2Soil | 4 | Chronic EMEG (pica child)4 |
| 100 | Chronic EMEG (child)4 | |||
| Endrin | 0.037 | 1992, #1Soil | 0.6 | Chronic EMEG (pica child) |
| 20 | Chronic EMEG (child) | |||
| Endrin Aldehyde | 0.032 | 1992, #2Soil | None | |
| Heptachlor Epoxide | 0.005 | 1992, #1Soil | 0.08 | CREG |
Sources: ATSDR, 1996a; Evergreen Engineering, 1996; and McGinnes Laboratories, 1992.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppm = parts per million.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. Comparison Value is for Endosulfan.
| Contaminant | Concentration Range1 (ppm)2 | Sample ID of Maximum Concentration | Concentration On-Site Residence (ppm) | Comparison Value 3 Value (ppm) Source | |
|---|---|---|---|---|---|
| Acenaphthylene | 0.053 J4 | SS09A | ND7 | None | |
| Carbon Disulfide | 0.004 J; 0.005 J | SS07B | 0.003 J | 200 | RMEG (pica child) |
| 5,000 | RMEG (child) | ||||
| Alpha - Chlordane | 0.0042 - 1 | SS03A | ND | 1 | Chronic EMEG (pica child)8 |
| 30 | Chronic EMEG (child)8 | ||||
| Gamma - Chlordane | 0.0044 - 1.1 | SS03A | ND | 1 | Chronic EMEG (pica child)8 |
| 30 | Chronic EMEG (child)8 | ||||
| Chlordane | 0.006 I10 - 14 | SS03A | ND | 1 | Chronic EMEG (pica child) |
| 30 | Chronic EMEG (child) | ||||
| Bis (2-ethylhexyl)phthalate | 0.470 - 6.2 J | SS03A | ND | 50 | CREG |
| 40 | RMEG (pica child) | ||||
| 1000 | RMEG (child) | ||||
| Captan | 0.003 | SS03B | ND | 300 | RMEG (pica child) |
| 7,000 | RMEG (child) | ||||
| Chrysene | 0.077 J - 0.19 J | SS09A | ND | 88 | RBCc |
| Flouranthene | 0.041 J - 0.38 | SS09A | ND | 80 | RMEG (pica child) |
| 2,000 | RMEG (child) | ||||
| Benzo (a) anthracene | 0.065 J - 0.2 J | SS09A | ND | 0.88 | RBCc |
| Benzo (b and/or k)flouranthene | 0.051 J - 0.24 J | SS09A | ND | 0.88 | RBCc [benzo(b)flouranthene] |
| 8.8 | RBCc [benzo(k)flouranthene] | ||||
| Benzo (g,h,i) perylene | 0.046 J; 0.099 J | SS09A | ND | None | |
| Benzo (a) pyrene | 0.050 J - 0.17 J | SS09A | ND | 0.1 | CREG |
| 4,4' - DDE | 0.0034 I - 0.36 | SS09A | ND | 2 | CREG |
| 4,4' - DDD | 0.0013 I - 0.069 J | SS03A | ND | 3 | CREG |
| 4,4' - DDT | 0.0014 I - 0.33 | SS03A | ND | 1 | RMEG (pica child) |
| 30 | RMEG (child) | ||||
| Indeno (1,2,3-cd)pyrene | 0.055 J; 0.11 J | SS09A | ND | 0.88 | RBCc |
| PCB - 1260 | 0.050 - 0.79 | SS02B | ND | 1 | RMEG (child) 9 |
| Phenanthrene | 0.23 J | SS09A | ND | None | |
| Pyrene | 0.047 J - 0.47 | SS09A | ND | 60 | RMEG (pica child) |
| 2,000 | RMEG (child) | ||||
| Total Xylenes | ND | -- | 0.002 J | 400 | Intermediate EMEG (pica child) |
| 10,000 | Intermediate EMEG (child) | ||||
| Aluminum | 19 - 3,000 | SS02A | 510 | 78,000 | RBCn |
| Arsenic | 0.7 I - 2.3 | SS09A | ND | 0.5 | CREG |
| 0.6 | Chronic EMEG (pica child) | ||||
| 20 | Chronic EMEG (child) | ||||
| Barium | 0.45 I - 12 | SS07A | 6.9 | 100 | RMEG (pica child) |
| 4,000 | RMEG (child) | ||||
| Cadmium | 0.28 I - 1.1 | SS10A | ND | 0.4 | Chronic EMEG (pica child) |
| 10 | Chronic EMEG (child) | ||||
| Calcium | 220 - 66,000 | SS06A | 87,000 | None | |
| Chromium | 0.51 I - 96.7 | SS10A | 3.6 | 10 | RMEG (pica child)5 |
| 300 | RMEG (child)5 | ||||
| Copper | 3.7 J - 130 J | SS07A | 7.6 J | 3,100 | RBCn |
| Iron | 110 - 2,900 | SS03A | 460 | 23,000 | RBCn |
| Lead | 0.7 J - 31 | SS03A | 3.1 J | 500 | EPA Action Level |
| Magnesium | 170 - 920 | SS07A | 330 | None | |
| Manganese | 2.1 J - 650 J | SS07A | 4.7 J | 10 | RMEG (pica child) |
| 300 | RMEG (child) | ||||
| 4,000 | RMEG (adult) | ||||
| Total Mercury | 0.19 | SS09A | ND | 4 | Intermediate EMEG (pica child)6 |
| 100 | Intermediate EMEG (child)6 | ||||
| Potassium | 18 - 150 | SS07A | 69 | None | |
| Selenium | 0.9 I; 1 I | SS02A | ND | 10 | Chronic EMEG (pica child) |
| 300 | Chronic EMEG (child) | ||||
| Silver | 0.19 I - 1.3 | SS06A | ND | 10 | RMEG (pica child) |
| 300 | RMEG (child) | ||||
| Sodium | 500 | SS06A | 990 | None | |
| Vanadium | 1.1 J - 3 J | SS07A | 2.5 J | 6 | Intermediate EMEG (pica child) |
| 200 | Intermediate EMEG (child) | ||||
| Zinc | 4.6 J - 290 J | SS07A | 12 J | 600 | Chronic EMEG (pica child) |
| 20,000 | Chronic EMEG (child) | ||||
| Cyanide | 0.61 J (2 hits) | SS03B | ND | 100 | Intermediate EMEG (pica child) |
| 3,000 | Intermediate EMEG (child) | ||||
Sources: EPA, 1998b; EPA, 1998d; EPA, 1998e; EPA, 1998f; and FDEP, 1998.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppm = parts per million.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. J = estimated value.
5. Comparison value is for hexavalent chromium.
6. Comparison value is for inorganic mercury.
7. ND = material was analyzed for but not detected (value below the minimum quantitation limit).
8. Comparison value is for chlordane.
9. Comparison value is for PCB - 1254.
10. I = value reported is less than the minimum quantitation limit, and greater than or equal to the minimum detection limit.
| Contaminant | Concentration Range1 (ppm)2 | Sample ID of Maximum Concentration | Comparison Value 3 Value (ppm) Source | |
|---|---|---|---|---|
| Bis (2-ethylhexyl)phthalate | 0.62 J 4 | SS08 | 50 | CREG |
| 40 | RMEG (pica child) | |||
| 1000 | RMEG (child) | |||
| Carbon Disulfide | 0.015 | SS01 | 200 | RMEG (pica child) |
| 5,000 | RMEG (child) | |||
| 4,4' - DDD | 0.00097 I6 | SS01 | 3 | CREG |
| 4,4' - DDE | 0.0019 I | SS01 | 2 | CREG |
| 4,4' - DDT | 0.0037 | SS01 | 2 | CREG |
| 1 | RMEG (pica child) | |||
| 30 | RMEG (child) | |||
| Aluminum | 310; 640 | SS01 | 78,000 | RBCn |
| Arsenic | 22.3; 43 J | SS01 | 0.5 | CREG |
| 0.6 | Chronic EMEG (pica child) | |||
| 20 | Chronic EMEG (child) | |||
| 200 | Chronic EMEG (adult) | |||
| Barium | 4.24 - 6.4 | SS08 | 100 | RMEG (pica child) |
| 4,000 | RMEG (child) | |||
| Calcium | 1,800; 8,100 | SS01 | None | |
| Chromium | 2.75 A7 - 53 | SS01 | 10 | RMEG (pica child)5 |
| 300 | RMEG (child)5 | |||
| Copper | 21 J; 83 J | SS01 | 3,100 | RBCn |
| Iron | 220; 610 | SS01 | 23,000 | RBCn |
| Lead | 2.1 A - 9.3 | SS01 | 500 | EPA Action Level |
| Magnesium | 170 | SS08 | None | |
| Manganese | 15 J; 68 J | SS08 | 10 | RMEG (pica child) |
| 300 | RMEG (child) | |||
| 4,000 | RMEG (adult) | |||
| Potassium | 34; 43 | SS08 | None | |
| Vanadium | 0.48 J; 1.3 J | SS01 | 6 | Intermediate EMEG (pica child) |
| 200 | Intermediate EMEG (child) | |||
| Zinc | 16; 57 J | SS08 | 600 | Chronic EMEG (pica child) |
| 20,000 | Chronic EMEG (child) | |||
Sources: EPA, 1998b; EPA, 1998d; EPA, 1998f; and FDEP, 1998.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppm = parts per million.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. J = estimated value.
5. Comparison value is for hexavalent chromium.
6. I = value reported is less than the minimum quantitation limit, and greater than or equal to the minimum detection limit.
7. A = value reported is the mean of two or more determinations.
| Contaminant | Sediment St. Lucie River Concentration Range (ppm)2 | Sediment Drainage Ditches Concentration Range1 (ppm) | Sediment Drainage Ditches Sample ID of Maximum Concentration | Comparison Value 3 Value (ppm) Source | |
|---|---|---|---|---|---|
| Atrazine | ND | 0.0027 I9 | SD01 | 70 | RMEG (pica child) |
| 2,000 | RMEG (child) | ||||
| Alpha - Chlordane | ND6 | 0.014 | SD04 | 0.5 | CREG7 |
| 1 | Chronic EMEG (pica child)7 | ||||
| 30 | Chronic EMEG (child)7 | ||||
| Gamma - Chlordane | ND | 0.016 | SD04 | 0.5 | CREG7 |
| 1 | Chronic EMEG (pica child)7 | ||||
| 30 | Chronic EMEG (child)7 | ||||
| Chlordane | 0.025 I | 0.058 - 0.44 | SD04 | 0.5 | CREG |
| 1 | Chronic EMEG (pica child) | ||||
| 30 | Chronic EMEG (child) | ||||
| Bis (2-ethylhexyl)phthalate | 1.3 | 4.8 | SD01 | 50 | CREG |
| 40 | RMEG (pica child) | ||||
| 1000 | RMEG (child) | ||||
| Chrysene | ND | 0.071 J4 - 0.120 I | SD04 | 88 | RBCc |
| Flouranthene | ND | 0.13 J - 0.150 I | SD04 | 80 | RMEG (pica child) |
| 2,000 | RMEG (child) | ||||
| Benzo (b and/or k)flouranthene | ND | 0.13 - 0.22 I | SD04 | 0.88 | RBCc [benzo(b)flouranthene] |
| 8.8 | RBCc [benzo(k)flouranthene] | ||||
| Benzo (a) pyrene | ND | 0.063 J | SD03 | 0.1 | CREG |
| 4,4' - DDD | 0.0014 I | 0.0023 I - 0.0024 I | SD02 | 3 | CREG |
| 4,4' - DDE | 0.0015 I | 0.0096 - 0.030 | SD02 | 2 | CREG |
| 4,4' - DDT | 0.012 | 0.0042 J; 0.0092 | SD02 | 2 | CREG |
| 1 | RMEG (pica child) | ||||
| 30 | RMEG (child) | ||||
| (3 and/or 4)methylphenol | 0.058 J | ND | -- | 100 | RMEG (pica child) (3-methylphenol) |
| 3,000 | RMEG (child) (3- methylphenol) | ||||
| 390 | RBCn (4-methylphenol) | ||||
| PCB - 1242 | 0.039 J | ND | -- | 1 | RMEG (child)8 |
| Pyrene | ND | 0.12 J - 0.18 I | SD04 | 60 | RMEG (pica child) |
| 2,000 | RMEG (child) | ||||
| Toluene | ND | 0.003 J; 0.0039 I | SD03 | 400 | RMEG (pica child) |
| 10,000 | RMEG (child) | ||||
| Aluminum | 230 | 300 - 4,000 | SD02 | 78,000 | RBCn |
| Arsenic | ND | 0.7 I - 1 I | SD04 | 0.5 | CREG |
| 0.6 | Chronic EMEG (pica child) | ||||
| 20 | Chronic EMEG (child) | ||||
| Barium | 1.1; 7.48 | 3.06 - 10 | SD02 | 100 | RMEG (pica child) |
| 4,000 | RMEG (child) | ||||
| Cadmium | ND | 0.25 I - 0.53 I | SD04 | 0.4 | Chronic EMEG (pica child) |
| 10 | Chronic EMEG (child) | ||||
| Calcium | 1,800 | 1,800 - 19,000 | SD01 | None | |
| Chromium | 1.2 - 4.06 | 2.1 J - 20.4 | SD04 | 10 | RMEG (pica child)5 |
| 300 | RMEG (child)5 | ||||
| Copper | 7.2 J | 5.9 J - 51 J | SD04 | 3,100 | RBCn |
| Iron | 2,700 | 480 - 3,100 | SD02 | 23,000 | RBCn |
| Lead | 1.8 - 2.0 I | 2.5 - 10 | SD04 | 500 | EPA Action Level |
| Magnesium | ND | 590; 640 | SD02 | None | |
| Manganese | 15 J | 2.4 J - 110 J | SD02 | 10 | RMEG (pica child) |
| 300 | RMEG (child) | ||||
| 4,000 | RMEG (adult) | ||||
| Potassium | 8.9 J | 14 - 52 | SD04 | None | |
| Silver | ND | 0.25 I | SD04 | 10 | RMEG (pica child) |
| 300 | RMEG (child) | ||||
| Vanadium | 0.78 J | 0.82 J - 8.9 J | SD02 | 6 | Intermediate EMEG pica child) |
| 200 | Intermediate EMEG (child) | ||||
| Zinc | 7.6 J | 4.4 J - 68 | SD04 | 600 | Chronic EMEG (pica child) |
| 20,000 | Chronic EMEG (child) | ||||
Sources: EPA, 1998b; EPA, 1998d; EPA, 1998e; EPA, 1998f; and FDEP, 1998.
Table Notes:
1. The values listed in this column indicate the range of detected concentrations for each contaminant.
2. ppm = parts per million.
3. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
4. J = estimated value.
5. Comparison value is for hexavalent chromium.
6. ND = material was analyzed for but not detected (value below the minimum quantitation limit).
7. Comparison value is for chlordane.
8. Comparison value is for PCB - 1254.
9. I = value reported is less than the minimum quantitation limit, and greater than or equal to the minimum detection limit.
| Contaminant | Concentration (ppb)1 | Comparison Value2 Value Source | |
|---|---|---|---|
| Chlordane | 8 | 6 | Chronic EMEG (child) |
| 20 | Chronic EMEG (adult) | ||
| 4,4' - DDD | 0.3 | 0.1 | CREG |
| 4,4' - DDE | 1.0 | 0.1 | CREG |
| 4,4' - DDT | 0.5 | 5 | RMEG (child) |
| 20 | RMEG (adult) | ||
| Dieldrin | 0.75 | 0.5 | Chronic EMEG (child) |
| 2 | Chronic EMEG (adult) | ||
| Endosulfan II | 0.4 | 20 | Chronic EMEG (child)3 |
| 70 | Chronic EMEG (adult)3 | ||
| Ethyl benzene | 8 | 1,000 | RMEG (child) |
| 4,000 | RMEG (adult) | ||
| 700 | MCL | ||
| p-Xylene | 2 | 520 | RBCn |
| m-Xylene | 11 | 6,000 | Intermediate EMEG (child) |
| 20,000 | Intermediate EMEG (adult) | ||
| o-xylene | 12 | 1,400 | RBCn |
Sources: ATSDR, 1996a; and Evergreen Engineering, 1996.
Table Notes:
1. ppb = parts per billion.
2. Please refer to Appendix D for an explanation of the comparison values used in this public health assessment.
3. Comparison value is for Endosulfan.
APPENDIX D -- COMPARISON VALUES
Comparison Values
ATSDR comparison values are media-specific concentrations that are considered to be safeunder default conditions of exposure. They are used as screening values in the preliminaryidentification of site-specific "contaminants of concern". The latter term should not bemisinterpreted as an implication of "hazard". As ATSDR uses the phrase, a "contaminant ofconcern" is merely a chemical substance detected at the site in question and selected by thehealth assessor for further evaluation of potential health effects. Generally, a chemical is selectedas a "contaminant of concern" because its maximum concentration in air, water, or soil at thesite exceeds one of ATSDR's comparison values.
However, it must be emphasized that comparison values are not thresholds of toxicity. Whileconcentrations at or below the relevant comparison value may reasonably be considered safe, itdoes not automatically follow that any environmental concentration that exceeds a comparisonvalue would be expected to produce adverse health effects. The whole purpose behind highlyconservative, health-based standards and guidelines is to enable health professionals torecognize and resolve potential public health hazards before they can become actual publichealth consequences. Thus, comparison values are designed to be preventive, rather thanpredictive, of adverse health effects. The probability that such effects will actually occurdepends, not on environmental concentrations alone, but on a unique combination of site-specific conditions and individual lifestyle and genetic factors that affect the route, magnitude,and duration of actual exposure.
Listed and described below are the various comparison values that ATSDR uses to select chemicals for further evaluation, as well as other non-ATSDR values that are sometimes used to put environmental concentrations into a meaningful frame of reference.
| CREG | = | Cancer Risk Evaluation Guides |
|---|---|---|
| MRL | = | Minimal Risk Level |
| EMEG | = | Environmental Media Evaluation Guides |
| IEMEG | = | Intermediate Environmental Media Evaluation Guide |
| RMEG | = | Reference Dose Media Evaluation Guide |
| RfD | = | Reference Dose |
| RfC | = | Reference Dose Concentration |
| RBC | = | Risk-Based Concentration |
| DWEL | = | Drinking Water Equivalent Level |
| MCL | = | Maximum Contaminant Level |
| CLHA | = | Child Longer Term Health Advisory |
Cancer Risk Evaluation Guides (CREGs) are estimated contaminant concentrations expected to cause no more than one excess cancer in a million persons exposed over a lifetime. CREGs are calculated from EPA's cancer slope factors, or cancer potency factors, using default values for exposure rates. However, neither CREGs nor CSFs can be used to make realistic predictions of cancer risk. The true risk is always unknown and may be as low as zero.
Minimal Risk Levels (MRL) are estimates of daily human exposure to a chemical (dosesexpressed in mg/kg/day) that are unlikely to be associated with any appreciable risk ofdeleterious noncancer effects over a specified duration of exposure. MRLs are calculated usingdata from human and animal studies and are reported for acute (< 14 days), intermediate (15-364 days), and chronic (> 365 days) exposures. MRLs are published in ATSDR ToxicologicalProfiles for specific chemicals.
Environmental Media Evaluation Guides (EMEGs) are concentrations that are calculatedfrom ATSDR minimal risk levels by factoring in default body weights and ingestion rates.
Intermediate Environmental Media Evaluation Guides (IEMEG) are calculated fromATSDR minimal risk levels; they factor in body weight and ingestion rates for intermediateexposures (those occurring for more than 14 days and less than 1 year).
Reference Dose Media Evaluation Guide (RMEG) is the concentration of a contaminant inair, water or soil that corresponds to EPA's RfD for that contaminant when default values forbody weight and intake rates are taken into account.
EPA's Reference Dose (RfD) is an estimate of the daily exposure to a contaminant unlikely tocause noncarcinogenic adverse health effects. Like ATSDR's MRL, EPA's RfD is a dose expressed in mg/kg/day.
Reference Concentrations (RfC) is a concentration of a substance in air that EPA considersunlikely to cause noncancer adverse health effects over a lifetime of chronic exposure.
Risk-Based Concentrations (RBC) are media-specific concentrations derived by Region III ofthe Environmental Protection Agency Region III from RfDs, RfC's, or EPA's cancer slopefactors. They represent concentrations of a contaminant in tap water, ambient air, fish, or soil(industrial or residential) that are considered unlikely to cause adverse health effects over alifetime of chronic exposure. RBCs are based either on cancer ("c") or noncancer ("n") effects.
Drinking Water Equivalent Levels (DWEL) are based on EPA's oral RfD and representcorresponding concentrations of a substance in drinking water that are estimated to havenegligible deleterious effects in humans at an intake rate of 2 L/day for life, assuming thatdrinking water is the sole source of exposure.
Maximum Contaminant Levels (MCLs) represent contaminant concentrations in drinkingwater that EPA deems protective of public health (considering the availability and economics ofwater treatment technology) over a lifetime (70 years) at an exposure rate of 2 liters of waterper day.
Child Longer Term Health Advisory (CLHA) represents the concentration of a substance indrinking water that would have no deleterious effect on a child exposed for up to 7 years.
Reference for Comparison Values:
Agency for Toxic Substances and Disease Registry. Health Assessment Guidance Manual.Atlanta: ATSDR, March, 1992.
APPENDIX E -- ATSDR METHODOLOGY
ATSDR Methodology
ATSDR evaluates contaminants detected in environmental media at the site and determineswhether an exposure to them has public health significance. ATSDR selects and discusses thecontaminants based upon the following factors:
- concentrations of contaminants on and off site;
- community health concerns; and
- comparison of on- and off-site concentrations with ATSDR health comparisonvalues for (1) noncarcinogenic endpoints and (2) carcinogenic endpoints.
ATSDR health comparison values are concentrations of contaminants that are media specific(e.g., water, air, or soil). The comparison values are considered to be safe under defaultconditions of exposure and are used as screening values in the preliminary identification of site-specific "contaminants of concern." The "contaminants of concern" are those contaminants thatwere detected above the screening comparison values and contaminants without comparisonvalues. However, the comparison values in the Tables in Appendix C are those that ATSDRconsiders to be the most appropriate, considering site-specific conditions of exposure, i.e.,specifically the duration of exposure (acute, intermediate or chronic) to the environmentalmedium in question most likely to prevail at the site. Please refer to the ToxicologicalEvaluation section and Appendix D for further clarification and description of the comparisonvalues used in this public health assessment.
Following the preliminary identification of site-specific "contaminants of concern", which aredescribed in the Environmental Contamination and Other Hazards section, ATSDR staff discussin the Pathways Analyses section whether nearby residents are exposed to contaminationmigrating from the site. If exposure to contamination is identified, the significance of thisexposure, with relation to adverse health effects, is discussed in the Toxicological Evaluationsection. ATSDR staff also address specific community concerns in the Community Health Concerns Evaluation section. Finally, based on the evaluations from all preceding sections of thepublic health assessment, ATSDR staff determine conclusions and prepare recommendations.
APPENDIX F -- ATSDR HEALTH CONSULTATION
The following section was not available in electronic format for conversion to HTML at the time of preparation of this document. To obtain a hard copy of the document, please contact:
Agency for Toxic Substances and Disease Registry
Division of Health Assessment and Consultation Branch, E-56
1600 Clifton Road NE, Atlanta, Georgia 30333
APPENDIX G -- ATSDR RESPONSE TO COMMENTS
ATSDR RESPONSE TO COMMENTS
This appendix contains a summary of the comments ATSDR received during the publiccomment period for the Loxahatchee Nursery public health assessment. Each comment waslogged and became part of the administrative record. Both the comments and ATSDR'sresponses are included in the text below. The comments have been numbered and are in italic with ATSDR's response directly below each comment. Personal identifiers and namesmentioned in the comments ATSDR received have not been included. Also, similar commentsreceived from different individuals have been combined into one comment.
1. We want this nursery resampled for the breakdown products of benlate along with itscontaminants, flusilazole and chlorothalonil. We want two sets of samples taken at the sametime and one set sent to an out-of-state laboratory of our choosing.
ATSDR is considering sampling at the Loxahatchee Nursery site and testing for the chemicalflusilazole. Please refer to the Community Health Concerns Evaluation section of this publichealth assessment, which has been modified from the public comment version to include moreinformation concerning flusilazole. Benlate's breakdown product, carbendazim, has beenanalyzed for in private wells, monitoring wells, soil, and sediment without being detected.Therefore, ATSDR does not recommend any further analyses for benlate or its breakdownproducts.
2. We want the pond and its fish to be tested.
Because ATSDR has no chemical-specific environmental data from the pond, the agency hasidentified the pond as a "data gap" and is considering sampling water and sediment from thepond.
3. Independent soil and water analysis should be conducted on the properties surroundingthe nursery.
At this time, ATSDR does not recommend any further residential soil sampling. Soil and wateranalyses have been performed on the properties surrounding the nursery. No contaminants atlevels of health concern were detected. Of note, the MCHD plans to periodically monitor thegroundwater quality of private potable water sources in the area.
4. We want a cancer survey of the residents of the surrounding area.
From ATSDR's perspective, an adverse health effect to be evaluated should be plausibly relatedto the release of hazardous substances from the site being investigated. ATSDR reviewedenvironmental data for the Loxahatchee Nursery site and did not identify any contaminants at levels of concern for increased cancer risk. Of note, in response to a request from thecommunity to review the rate of cancer, the MCHD plans to review cancer incidence data. Formore information on this issue, please contact the MCHD directly.
5. We would like a federal investigation before a grand jury into the matter of federal andstate agencies collaborating with DuPont to withhold crucial life and health information fromthe people of the United States and the State of Florida.
This request is out of the purview of ATSDR's activities at the site.
6. In Appendix G, Health Consultation, it stated that the resident that had a high level of1,2-dichloropropane was receiving bottled water and the report made recommendation #5 thatmore information is needed to stop exposure. This well was fitted with a charcoal filter almostimmediately after the contamination was found. When ATSDR investigators visited the site, thefilter was installed on the well. This affects section B on page 4.
The information referred to in this comment pertains to recommendations ATSDR made in ourSeptember 1996 health consultation. These are not our current recommendations which arefound in the Recommendation section of this public health assessment. To avoid confusion,ATSDR has modified section B to only state that the 1996 health consultation recommendedadditional sampling activities. Information contained in the copy of the 1996 health consultationfound in Appendix G cannot be modified as it is considered a released, final document.
7. Several sections of the public health assessment reference historical use of 1,2-dichloropropane as a soil fumigant at the nursery. I have spoken with all owners or operatorsfrom 1944 to 1992, none of whom report ever using this chemical on the premises. They diduse methyl bromide as a nematocide.
ATSDR thanks the commentor for this information. It was reported to ATSDR that 1,2-dichloropropane was used on site and the agency included this information in the publiccomment release of the document. All references to the use of 1,2-dichloropropane on site havebeen removed from the main text of the final release public health assessment because thereports of its use are conflicting. Of note, not stating the source of the 1,2-dichlorpropane in thepublic health assessment does not affect ATSDR's conclusions and recommendations regardingthis site. Further, in the Community Health Concerns Evaluation section , ATSDR states that1,2-dichloropropane has not been detected in on-site monitoring well, soil, or sediment sampleswhich suggests the nursery is not the source.
8. The Background section reports that pesticides were applied via injection through theirrigation system. Fertilizers were occasionally applied in this fashion, but pesticides were not.
The sentence in question has been modified to indicated that fertilizers were delivered to theplants through the nursery's irrigation system.
9. The Background section reports that the nursery was developed on filled wetlands. Thisis not true. The topography, except for beds graded up from original grade, remains the sameas when the land was cleared in 1944. The same sentence also indicates that a tile drainagesystem exists beneath the site.
Based on this information, ATSDR has deleted the sentence in question from the main text ofthe public health assessment. Deleting this sentence does not alter ATSDR's conclusions andrecommendations regarding this site.
10. The report indicates a septic tank system is present on site. Was this located during theMarch 1998 sampling event? If so, why was the septic tank system not sampled?
The septic tank sy
APPENDIX G -- ATSDR RESPONSE TO COMMENTS
ATSDR RESPONSE TO COMMENTS
This appendix contains a summary of the comments ATSDR received during the publiccomment period for the Loxahatchee Nursery public health assessment. Each comment waslogged and became part of the administrative record. Both the comments and ATSDR'sresponses are included in the text below. The comments have been numbered and are in italic with ATSDR's response directly below each comment. Personal identifiers and namesmentioned in the comments ATSDR received have not been included. Also, similar commentsreceived from different individuals have been combined into one comment.
1. Â Â Â We want this nursery resampled for the breakdown products of benlate along with itscontaminants, flusilazole and chlorothalonil. We want two sets of samples taken at the sametime and one set sent to an out-of-state laboratory of our choosing.
ATSDR is considering sampling at the Loxahatchee Nursery site and testing for the chemicalflusilazole. Please refer to the Community Health Concerns Evaluation section of this publichealth assessment, which has been modified from the public comment version to include moreinformation concerning flusilazole. Benlate's breakdown product, carbendazim, has beenanalyzed for in private wells, monitoring wells, soil, and sediment without being detected.Therefore, ATSDR does not recommend any further analyses for benlate or its breakdownproducts.
2. Â Â Â We want the pond and its fish to be tested.
Because ATSDR has no chemical-specific environmental data from the pond, the agency hasidentified the pond as a "data gap" and is considering sampling water and sediment from thepond.
3. Â Â Â Independent soil and water analysis should be conducted on the properties surroundingthe nursery.
At this time, ATSDR does not recommend any further residential soil sampling. Soil and wateranalyses have been performed on the properties surrounding the nursery. No contaminants atlevels of health concern were detected. Of note, the MCHD plans to periodically monitor thegroundwater quality of private potable water sources in the area.
4. Â Â Â We want a cancer survey of the residents of the surrounding area.
From ATSDR's perspective, an adverse health effect to be evaluated should be plausibly relatedto the release of hazardous substances from the site being investigated. ATSDR reviewedenvironmental data for the Loxahatchee Nursery site and did not identify any contaminants at levels of concern for increased cancer risk. Of note, in response to a request from thecommunity to review the rate of cancer, the MCHD plans to review cancer incidence data. Formore information on this issue, please contact the MCHD directly.
5. Â Â Â We would like a federal investigation before a grand jury into the matter of federal andstate agencies collaborating with DuPont to withhold crucial life and health information fromthe people of the United States and the State of Florida.
This request is out of the purview of ATSDR's activities at the site.
6. Â Â Â In Appendix G, Health Consultation, it stated that the resident that had a high level of1,2-dichloropropane was receiving bottled water and the report made recommendation #5 thatmore information is needed to stop exposure. This well was fitted with a charcoal filter almostimmediately after the contamination was found. When ATSDR investigators visited the site, thefilter was installed on the well. This affects section B on page 4.
The information referred to in this comment pertains to recommendations ATSDR made in ourSeptember 1996 health consultation. These are not our current recommendations which arefound in the Recommendation section of this public health assessment. To avoid confusion,ATSDR has modified section B to only state that the 1996 health consultation recommendedadditional sampling activities. Information contained in the copy of the 1996 health consultationfound in Appendix G cannot be modified as it is considered a released, final document.
7. Â Â Â Several sections of the public health assessment reference historical use of 1,2-dichloropropane as a soil fumigant at the nursery. I have spoken with all owners or operatorsfrom 1944 to 1992, none of whom report ever using this chemical on the premises. They diduse methyl bromide as a nematocide.
ATSDR thanks the commentor for this information. It was reported to ATSDR that 1,2-dichloropropane was used on site and the agency included this information in the publiccomment release of the document. All references to the use of 1,2-dichloropropane on site havebeen removed from the main text of the final release public health assessment because thereports of its use are conflicting. Of note, not stating the source of the 1,2-dichlorpropane in thepublic health assessment does not affect ATSDR's conclusions and recommendations regardingthis site. Further, in the Community Health Concerns Evaluation section , ATSDR states that1,2-dichloropropane has not been detected in on-site monitoring well, soil, or sediment sampleswhich suggests the nursery is not the source.
8. Â Â Â The Background section reports that pesticides were applied via injection through theirrigation system. Fertilizers were occasionally applied in this fashion, but pesticides were not.
The sentence in question has been modified to indicated that fertilizers were delivered to theplants through the nursery's irrigation system.
9. Â Â Â The Background section reports that the nursery was developed on filled wetlands. Thisis not true. The topography, except for beds graded up from original grade, remains the sameas when the land was cleared in 1944. The same sentence also indicates that a tile drainagesystem exists beneath the site.
Based on this information, ATSDR has deleted the sentence in question from the main text ofthe public health assessment. Deleting this sentence does not alter ATSDR's conclusions andrecommendations regarding this site.
10. Â Â Â The report indicates a septic tank system is present on site. Was this located during theMarch 1998 sampling event? If so, why was the septic tank system not sampled?
The septic tank system was not located during the March 1998 sampling event. ATSDRbelieves the septic tank is used by the on-site residence; however, ATSDR was not able find anydocumentation to indicate the septic tank's location or use.
11. Â Â Â Pages 6 and 10 of the report appear to contradict the depth to the water table beneaththe site.
ATSDR does not believe the sentences in question contradict one another. Page 6 states, "Inthis area, the water table is encountered at 8 feet or less below ground surface." Page 10 thengoes on to state that, during the March 1998 sampling event, groundwater was encountered at1.5-2 feet below land surface. ATSDR does not see a contradiction with stating thatgroundwater in the area is encountered at 8 feet or less , because 1.5-2 feet falls within thepreviously defined range. The water table in this area fluctuates depending on the season.
12. Â Â Â In the Pathway Analysis section, the document reports one Completed ExposurePathway beginning with 1,2-dichloropropane applied at the nursery and ending with it in adrinking water well. As this chemical was not used on site, it originated elsewhere. In fact, thehigh relative concentration in the one well, which is located in a drainage swale north of thenursery, along with failure to find 1,2-dichloropropane anywhere else within the nursery orsurrounds, argues against the nursery being the source. It is much more likely that a localhomeowner used this chemical as a degreaser, which is one on its known applications, anddisposed of it in the drainage swale upgradient of the well in question.
As stated in comment number 7, ATSDR has removed references to the use of 1,2-dichloropropane on-site because the Agency has received conflicting information on this issue.However, a human exposure pathway can be "complete" even if the source of contamination is not known, so exposure to 1,2-dichloropropane remains in the public health assessment as acompleted exposure pathway.
13. Â Â Â The report states that three private wells had trace levels of 1,2-dichloropropane.However, trace levels of this solvent have been found in five private wells.
The draft public health assessment contained private well data sampled from 1992 to July 1996.Based on this comment, ATSDR requested updated data from MCHD. Additional data fromAugust to December 1996 were obtained and are included in this final public health assessment.Two additional wells were found to contain 1,2-dichloropropane at trace levels and the maintext of this document has been modified to indicate five wells instead of three.
14. Â Â Â The Martin County Public Health Unit (MCPHU) is now the Martin County HealthDepartment (MCHD).
ATSDR thanks the commentor for this information which has been added to the final publichealth assessment text in the Public Health Actions section.
15. Â Â Â The pond was sampled by FDEP and two 7-day chronic static-renewal definitivetoxicity bioassays were performed on the samples collected. The samples did not demonstratechronic toxicity to the test species. Although the samples had exceeded normal holding times,toxicants were expected to be stable.
ATSDR thanks the commentor for this information. However, because chemical-specificenvironmental sampling data (i.e., for surface water or sediment) have not been collected for thepond, ATSDR recommends additional characterization of the pond as indicated in theRecommendation section of the public health assessment.
16. Â Â Â How can LN-SS08, LN-SS01, and LN-TW01 be considered background or off site asthey appear to be on the triangular parcel of land that is part of the Loxahatchee Nursery?This parcel of land was a part of the nursery operations and did have ornamentals on it.
As indicated in several places in the Environmental Contamination and Other Hazards section ofthis public health assessment, ATSDR did not consider the sampling locations mentioned in thiscomment to reflect "background" conditions. Please refer to the main text for furtherclarification of the background issue. Additionally, references to "on -site" and "off site" inpublic health assessments are arbitrary boundaries created by ATSDR staff for the purpose ofdelineating data and evaluating human exposure pathways. The parcel in question is across thestreet from where primary nursery operations occurred. The parcel is directly adjacent to thecounty park. The data reports that ATSDR received indicated this parcel was outside ofproperty boundaries. For this public health assessment, ATSDR assumed that off-site exposures would occur more frequently than on-site exposures. For these reasons, ATSDR staff includedsampling data from the locations mentioned in this comment with the "off-site" data.
17. Â Â Â In the Community Health Concerns Evaluation section of the report, ATSDR states that"levels detected of these metals fall within observed ranges for metals in eastern United Statessoil and are probably not due to nursery activities." A more recent report (Ma et al, 1997) onFlorida soils suggests that the arsenic level at LN-SS01 and the chromium level at LN-SS10Aare indicative of anthropogenic sources.
ATSDR reviewed the report (Ma et al, 1997) the commentor graciously provided. The reportconcluded that background values of most metals in 40 Florida soils were lower than theaverage of United States soils. ATSDR has deleted the sentence in question from the main textof the public health assessment.
18. Â Â Â The sediment analytical results revealed exceedances of the Sediment GuidanceCriteria for chlordane; 4,4'-DDD; 4,4'-DDE; 4,4'-DDT; chrysene; flouranthene; and pyrene.The soil analytical results revealed exceedances of the Florida Soil Cleanup Goals forresidential direct exposure for arsenic in several samples. The soil analytical results revealedexceedances of the Florida Soil Cleanup Goals for residential direct exposure and leachabilityfor chlordane in two samples. The soil analytical results revealed exceedances of the FloridaSoil Cleanup Goals for leachability for chromium and dieldrin in one sample each. Thegroundwater analytical results revealed exceedances of the Florida Drinking Water Standardsfor 1,2-dichloropropane in one private well. As discussed in the text of ATSDR's report, thiswell has been fitted with a carbon filter which removes the contaminant from the water.
ATSDR thanks the commentor for this information. As stated in the main text, ATSDRconcluded that under site-specific conditions of exposure, none of the contaminants detected insoil or sediment are likely to pose a hazard to public health. ATSDR understands that if the siteis to be developed residential, certain criteria and cleanup goals set by the state of Florida mustbe met; however, as a nonregulatory agency, commenting on the cleanup goals set by the stateare out of the purview of this public health assessment.


